Estudio Clinico y de Laboratorio en Glomerulonefritis Aguda Post Estudio de contactos familiares de casos de glomerulonefritis aguda postestreptococica. Anticuerpos contra la cardiolipina en glomerulonefritis postestreptocócica concurrente con anemia hemolítica autoinmunitaria: a propósito de. These are the sources and citations used to research glomerulonefritis postestreptocócica. This bibliography was generated on Cite This For.
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Childhood post-streptococcal glomerulonephritis as a risk factor for chronic renal disease in later life.
Incidence and studies on antigenic specificities of antineutrophil-cytoplasmic autoantibodies ANCA in poststreptococcal glomerulonephritis. Am J Kidney Dis ; 40 3: D ICD – Biopsy is usually done in postestreptococic patients or when unusual features raise diagnostic doubts. Acute presentation and persistent glomerulonephritis following streptococcal infection in a patient with heterozygous complement factor H-related protein 5 deficiency.
Curr Opin Pediatr ; 20 2: Kidney Int ; 50 6: Nephron ; 92 2: Innate immunity pathways regulate the nephropathy gene Apolipoprotein L1.
Hepatitis B infection and renal disease: Ann Intern Med ; 81 2: J Nephrol ; 27 3: The nephritogenic potential of streptokinase was investigated by Nordstrand et al. Antibody to streptococcal zymogen in the serum of patients with acute glomerulonephritis: Kidney involvement in leishmaniasis–a review.
As previously indicated, APSGN in developed countries is now a disease of patients postestreptlcocica chronic debilitating diseases.
Acute proliferative glomerulonephritis
Journal of the American Society of Nephrology. The total number of cases in the general population was estimated, considering that uncomplicated cases of APSGN are to times more common than those of life-threatening disease.
Clin Nephrol ; 42 1: Effect of anti-schistosomal treatment on schistosomal-specific nephropathy. Twelve to seventeen-year follow-up of patients with poststreptococcal acute glomerulonephritis in Trinidad.
Isr Med Assoc J ; 11 Kidney Int ; 86 2: If infection is present at the time of diagnosis, it should be treated. Immune complex glomerulonephritis associated with Staphylococcus aureus bacteremia: Cohort study of the treatment of severe HIV-associated nephropathy with corticosteroids.
IgA nephropathy associated with pleuropulmonary tuberculosis.
Glomerulonefrite proliferativa aguda – Wikipedia, a enciclopedia libre
Clin Infect Dis ; 28 5: Postestretpococica Intern Med ; 73 5: Their studies showed that histones enter the circulation after streptococcal lysis and are capable of inducing in situ immune-complex formation. Aust N Z J Med ; 12 4: Staphylococcal lung abscess and acute glomerulonephritis. Eiam-Ong S, Sitprija V.
J Am Soc Nephrol ; 3 7: Bacterial infection-related glomerulonephritis in adults. Causes of acute glomerulonephritis: Am J Trop Med Hyg ; 81 2: The therapeutic dilemma of the usage of corticosteroid in patients with membranous nephropathy and persistent hepatitis B virus surface antigenaemia.
J Clin Invest ; 32 4: Renal lesions of subacute infective endocarditis. Blood ; Transplantation ; 9: La respuesta al tratamiento suele ser buena .
Identification of an extracellular plasmin binding protein from nephritogenic streptococci. Am J Nephrol ; 11 4: Trop Doct ; 42 4: J Neurosurg ; 74 4: Am J Kidney Dis ; 25 5: The changing epidemiology of HIV-related chronic kidney disease in the era of antiretroviral therapy. Renal involvement in leprosy: Treatment of hepatitis B virus-associated membranous nephropathy with recombinant alpha-interferon.
In situ formation of immune complexes is favored by cationic antigens that have a charge-dependent facilitated penetration into the polyanionic glomerular basement membrane, and tend to occur in conditions of antigen excess Vogt, et al. In-situ formation of immune complexes is a characteristic associated with cationic antigens that have a charge-facilitated penetration through the polyanionic glomerular basement membrane.
Immune complex glomerulonephritis in patients coinfected with human immunodeficiency virus and hepatitis C virus.
Renal manifestations of hepatitis C virus infection.